Non-arrhythmic causes of sudden death: A comprehensive review
نویسندگان
چکیده
منابع مشابه
Sudden adult death syndrome and other non-ischaemic causes of sudden cardiac death.
OBJECTIVE To evaluate non-atherosclerotic cardiac deaths in the UK population aged over 15 years including elderly patients and to highlight the concept of the structurally normal heart in sudden death. METHODS Pathological data were collected prospectively for sudden adult deaths referred by UK coroners. RESULTS 453 cases of sudden death from 1994 to 2003 (278 men (61.4%) and 175 women (38...
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Recent findings The primary purpose of the preparticipation examination is to detect the cardiovascular disorders known to cause sudden cardiac arrest in the athlete. Studies demonstrate that the traditional history and physicalbased examination has a limited sensitivity, does not detect the majority of athletes with at-risk conditions, and may provide false reassurance for athletes with disord...
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Athough it is difficult to determine the precise number, the range for the number of sudden cardiac deaths (SCDs) per year in the United States alone has been reported from 184 000 to 462 000,1 with estimates that 50% to 70% are due to tachyarrhythmic mechanisms. Regardless of where within this range the true number lies, this represents a large epidemiological problem that warrants serious att...
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Sudden cardiac death refers to unexpected death attributable to a cardiac cause occurring within 1 h of the onset of symptoms (NICE, 2006). It often results from cardiac arrhythmias and is a major worldwide cause of morbidity and mortality. Arrhythmias account for 180,000 to 250,000 deaths per year in the United States and ∼70,000 deaths per year in the United Kingdom (NICE, 2006; Chugh et al.,...
متن کاملMitochondria oxidative stress, connexin43 remodeling, and sudden arrhythmic death.
BACKGROUND Previously, we showed that a mouse model (ACE8/8) of cardiac renin-angiotensin system activation has a high rate of spontaneous ventricular tachycardia and sudden cardiac death secondary to a reduction in connexin43 level. Angiotensin-II activation increases reactive oxygen species (ROS) production, and ACE8/8 mice show increased cardiac ROS. We sought to determine the source of ROS ...
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ژورنال
عنوان ژورنال: Progress in Cardiovascular Diseases
سال: 2019
ISSN: 0033-0620
DOI: 10.1016/j.pcad.2019.05.004